Last edited by Moogushura
Wednesday, August 12, 2020 | History

4 edition of Protection of the ischemic myocardium found in the catalog.

Protection of the ischemic myocardium

cardioplegia

by David J. Hearse

  • 369 Want to read
  • 5 Currently reading

Published by Raven Press in New York .
Written in English

    Subjects:
  • Cardiac arrest, Induced.,
  • Heart -- Preservation.,
  • Ischemia.,
  • Coronary disease -- Surgery.,
  • Heart arrest, Induced.

  • Edition Notes

    Includes bibliographical references and index.

    StatementDavid J. Hearse, Mark V. Braimbridge, Per Jynge.
    ContributionsBraimbridge, M. V., Jynge, Per.
    Classifications
    LC ClassificationsQP114.A75 H4
    The Physical Object
    Paginationxii, 420 p. :
    Number of Pages420
    ID Numbers
    Open LibraryOL4431025M
    ISBN 100890044236
    LC Control Number79064428

    Nayler WG: Protection of the myocardium against post- ischemic reperfusion damage. The combined effect of hy- pothermia and nifedipine. J Thorac Cardiovasc Surg , Magovern GJ Jr, Flaherty JT, Gott VL, et al: Failure of blood cardioplegia to protect myocardium at lower temperature. Circulation 66(suppl 2), In addition, myocardial dysfunction is a frequent sequel of perinatal asphyxia, resulting from hypoxic-ischemic damage to the myocardium. It can lead to decreased perfusion, tachycardia, hypotension, and need for inotropic support [20,21]. As a consequence, hemodynamic impairment can develop and the myocardium may suffer additional ischemic Author: Fabio Carmona, Karina M. Mata, Marcela S. Oliveira, Simone G. Ramos.

      Myocardial injury after surgery Ventricular hypertrophyVentricular hypertrophy Pre-ischemic energy depletionPre-ischemic energy depletion Length of the ischemic intervalLength of the ischemic interval Incomplete myocardial protectionIncomplete myocardial protection Ventricular distention (failure to vent the LA adequately) Retraction and. A critical review of the most up-to-date research on purines and myocardial protection. The role of purines in reversible `myocardial stunning' and irreversible (myocardial infarction) ischemic injury, ventricular arrhythmias, and ischemic preconditioning is discussed in detail, by experts. All.

    Start studying ICDCM DZ of the Circulatory System Chapter 28 Nelly Text Book. Learn vocabulary, terms, and more with flashcards, games, and other study tools.   Lipid peroxidation is particularly active during reperfusion of ischemic myocardium and/or brain cells. The source of free radicals is not clear, but may include neutrophils infiltrating the ischemic myocardium, myocardial tissue, and endothelial cells. 12, 25 Carvedilol inhibits lipid peroxidation in myocardial cell membranes initiated by Cited by:


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Protection of the ischemic myocardium by David J. Hearse Download PDF EPUB FB2

Find many great new & used options and get the best deals for Protection of the Ischemic Myocardium: Cardioplegia by David J. Hearse, M.

Brainbridge and P. Jynge (, Book, Other) at the best online prices at eBay. Free shipping for many products. Additional Physical Format: Online version: Hearse, David J. Protection of the ischemic myocardium. New York: Raven Press, © (OCoLC) Myocardial Protection.

Magnesium also prevents the influx of sodium into the postischemic myocardium, which is exchanged for calcium during reperfusion.

syndrome can occur after any operation requiring cardiopulmonary bypass and cardioplegic arrest from either inadequate myocardial protection, long ischemic time.

Get this from a library. Myocardial ischemia: mechanisms, reperfusion, protection. [M Karmazyn;] -- Myocardial ischemia and subsequent reperfusion of the ischemic myocardium represent complex phenomena encompassing numerous physiological processes.

This book aims at enhancing our understanding of. Myocardial ischemia and subsequent reperfusion of the ischemic myocardium represent complex phenomena encompassing numerous physiological processes.

This book aims at enhancing our understanding of these processes and stresses recent important developments in this very active area of research. MJOS, O.: Effect of reduction of myocardial free fatty acid metabolism relative to that of glucose on the ischemic injury during experimental coronary artery occlusion in dogs, experimental and clinical aspects on preservation of the ischemic myocardium.

Hjalmarson, L. Werko, Lindgren and Soner, Sweden, Molndal, pp. 29–34 Google Cited by: 6. Abenschein DR, Tacker WA, Babbs CF: Protection of ischemic myocardium by whole body hypothermia after coronary artery occlusion in dogs.

Am Heart JSaito Y, Yasutomi N, Nomura Y, et al: Protection of ischemic myocardium by whole body hypothermia after acute coronary occlusion in the closed chest by:   And despite the complexity of this subject, the book is written with exemplary clarity.

Myocardial protection stems from two lines of thought: the concept of the reversibility of tissue injury after reperfusion, and the supposition that cellular metabolism can be manipulated to salvage cells that might otherwise have : Alan T.

Marty. It was written by a basic scientist, a clinical investigator, and a clinical cardiac surgeon, each of whom imparts his own perspective on the protection of the ischemic myocardium.

The book. Cellular protection during myocardial ischemia: the development and characterization of a procedure for the induction of reversible ischemic arrest. Circulation ; 54 (2): – Melrose, DG, Dreyer, B, Bentall, HH, et al.

Elective cardiac arrest. This book is a thorough description of the current state of knowledge of the mechanisms of heat shock proteins induced cardiac protection at the cellular and molecular level, the controversies in this growing field and the potential of treating ischemic heart disease with overexpression of heat shock proteins in by: 2.

Oxygen demand in regionally ischemic myocardium is 70% that in nonischemic working myocardium and exceeds that of globally ischemic myocardium by a factor of 4 ( mL O 2 /min per g of tissue vs.

mL O 2 /min per g of tissue). Hence, the regionally ischemic myocardium has a greater mismatch between energy supply and demand than. Myocardial ischemic injury and protection Article (PDF Available) in Experimental and clinical cardiology 9(4) February with 23 Reads How we measure 'reads'. It is, in effect, ischemic myocardium supplied by a narrowed coronary artery in which ischemic cells remain viable, but contraction is chronically depressed.

Here, the contractile function of the involved myocardium can be partially or even totally restored by improving the coronary blood flow or reducing the oxygen demand of the : Yash Vaidya, Shaelyn Cavanaugh, Amit Dhamoon.

Pathophysiology of myocardial infarction. Protection by ischemic pre- and postconditioning Article Literature Review in Herz 33(2) April with 43 Reads.

Michael Galagudza (February 29th ). Cardiac Protection with Targeted Drug Delivery to Ischemic-Reperfused Myocardium, Novel Strategies in Ischemic Heart Disease, Umashankar Lakshmanadoss, IntechOpen, DOI: / Available from:Author: Michael Galagudza.

cells at border of ischemic zone. 2 important time periods for someone having an MI. Set up for ventricular fibrillation. Cells are firing off too early. Creates gradient of excitability.

Injured myocardium stretches differently from the healthy myocardium. That injury actually causes electrical signal, which is the trigger. Ischemic preconditioning (IPC) is an experimental technique for producing resistance to the loss of blood supply, and thus oxygen, to tissues of many types.

In the heart, IPC is an intrinsic process whereby repeated short episodes of ischaemia protect the myocardium against a subsequent ischaemic insult. It was first identified in by Murry et group exposed MeSH: D This book is a thorough description of the current state of knowledge of the mechanisms of heat shock proteins induced cardiac protection at the cellular and molecular level, the controversies in this growing field and the potential of treating ischemic heart disease with overexpression of heat shock proteins in : $   Phase 4: the prevention of lethal myocardial reperfusion injury.

Many interventions to prevent or diminish lethal myocardial reperfusion injury have been studied. 25 Two are particularly interesting and have shown some promise, both in preclinical studies as well as in small, but intriguing, proof of principle clinical trials. The first is an extension of the principle of Cited by:.

His research focuses on: (i) elucidating the mechanisms of ischemic- pharmacologic- and exercise-induced preconditioning by using the ischemia/reperfusion model in genetically engineered animals, (ii) studying protection of ischemic myocardium by using gene and/or cell therapy, and (iii) elucidating adaptations to ischemia/reperfusion injury in.attract neutrophils to ischemic/reperfused tissue.

Preconditioning can only delay cell death ; ineff if sustained ischemic insult gt 3 hrs ; Preconditioning failed to protect the mid and subepicardial myocardium; Second phase of protection req 24 hours to appear sustained for up to 72 hours. Second window of protection (SWOP), late [email protected]{osti_, title = {Cardiac progenitor-derived exosomes protect ischemic myocardium from acute ischemia/reperfusion injury}, author = {Chen, Lijuan and Cardiovascular Disease, Internal Medicine, University of Cincinnati, Albert Sabin Way, Cincinnati, OH and Wang, Yingjie and Internal Medicine of Traditional Chinese.